POTD: Dog Bites

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In celebration of the Year of the Dog, we wanted to cover management of dog bites.

According to CDC data from 2015, there are approximately 4.5 million dog bites per year in the United States with 1 out of 5 requiring medical attention. The wounds tend to be crush injuries with a greater risk of underlying fracture due to the strength of the dog’s jaws. Pay attention to distal neurovascular status, tendon involvement, joint violation and the presence of foreign bodies. A low threshold to x-ray is valuable. These wounds should be debrided and cleaned with well pressured irrigation.

Classically tested, the most common pathogen that creates infection in wounds is Pasteurella Canis (and other Pasteurella species). Immunosuppressed, alcoholics, smokers or asplenic patients should raise concern for Capnocytophagia canimorsus (a gram-negative rod) that causes particularly devastating illness with meningitis and septic shock reported.

Antibiotic prophylaxis/treatment of choice is with amoxicillin-clavulanate 875/125mg twice a day for 10-14 days. Other bacteria of interest include staphylococci, streptococci, and anaerobes. Remember this isn't your run of the mill cellulitis, cephalexin will not cut it.

Repairing these bites has been a subject of debate. REBEL-EM did a great job covering the myths for these wounds with two of the major studies. (link below)

Using 3-7% as a normal wound infection rate for all lacerations, the thought is that you can attempt a closure on some of these wounds for cosmesis. Good indications for closure would be a clean appearing wound that can receive significant irrigation that is <8 hours old. Wounds greater than 8 hours old had greater than a 20% chance of infection if closed in a study by Paschos et al.

Well vascularized areas perform better with closure – the face/scalp. We use non-absorbable sutures and no buried sutures to reduce the burden of foreign bodies present – minimizing infectious risk.

Tetanus should be given to patients suffering dog bites if they have not received it in the past 5 years. Rabies vaccination + rabies immunoglobulin should be considered for dog bites occurring in the USA from dogs that cannot be monitored and/or are unvaccinated. People previously vaccinated against the rabies virus do not need the immunoglobulin but can take part in the 0, 3, 7, 14 series tailored per local infectious disease recommendations.

Dog bites that return to the ED with infection should be cultured (with peripheral smear added for patients at risk of Capnocytophagia). The area should be imaged to assess the integrity of the bone.

Happy year of the dog!

Read More

Centers for Disease Control and Prevention. Preventing Dog Bites. http://www.cdc.gov/features/dog-bite-prevention/index.html. May 18, 2015.

Paschos NK et al. Primary closure versus non-closure of dog bite wounds. A radomised controlled trial. Injury 2014 45(1): 237-40PubMed ID: 23916901

Medeiros IM, Saconato H. Antibiotic prophylaxis for mammalian bite (Review). Cochrane Database of Systematic Reviews 2008 (3); PubMed ID: 11406003

Butler T et al. Capnocytophaga canimorsus: an emerging cause of sepsis, meningitis, and post-splenectomy infection after dog bites. Eur J Clin Microbiol Infect Dis. 2015 34(7): 1271-80. PubMed ID: 25828064

http://rebelem.com/myths-management-dog-bites/


Trauma Tuesday: Handlebar Injuries

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Case: Pediatric patient arrives after being overzealous on his bicycle with the following wound.

The first thing you do is recognize the sign. The second thing you do is ask to look at a video of what happened.

What we see above is the handlebar sign. It can present either as a longitudinal pattern of the bicycle handlebars strike the abdomen in collision or it may be a circular wound from the end of the handlebar impaling the abdomen.

Management:

An estimated 10% of bicycle injuries are related to contact with handlebars. Hemodynamically unstable patients should raise consideration for injury to the IVC or other abdominal vascular structures.

Your trauma survey places a lot of importance on the chest xray which can show diaphragmatic rupture or significant viscous perforation early on. Early laparotomy should occur in unstable patients, patients with significant peritonitis, or free air on x-ray.

Patients with isolated injuries to the abdomen, a negative FAST, normal labs (including LFTs/lipase/UA), and clinical improvement over 24 hours are safe for discharge.

Persistent LUQ pain that radiates to left shoulder during serial exams will generally require further investigation including advanced imaging like CT with IV contrast. If bilious vomiting ensues 24-48 hours after injury, consider a duodenal hematoma as hollow viscous injuries are rarely seen on CT scans.

Splenic injuries require serial hemoglobin/hematocrits, serial abdominal exams, and bed rest. Grades 1-4 are non-operative per American Pediatric Surgical Association. Splenectomy is rarely required though vaccination for encapsulated bacteria should be performed in the setting of severe injuries.

Read More: Gutierrez IM, Ben-Ishay O, Mooney DP. Pediatric thoracic and abdominal trauma. Minerva Chir 2013;68:263-274.

Puskarich MA, Marx JA: Abdominal Trauma, in Marx JA, Hockberger RS, Walls RM, et al (eds): Rosen’s Emergency Medicine: Concepts and Clinical Practice, ed 8. St. Louis, Mosby, Inc., 2014, (Ch) 46:p 459-478,

Teisch LF, Allen CJ, Tashiro J, et al. Injury patterns and outcomes following pediatric bicycle accidents. Pediatr Surg Int 2015;31:1021-1025.


Headache and nausea after a night out on the town: Just a hangover? Or a "can't miss" diagnosis?

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It’s In-Service season, people! Let’s review the essentials of carbon monoxide poisoning. And before you start with your “Ugh, really? How many times do we have to—“, let me cut you off and have you gimme the full list of indications for hyperbaric oxygen therapy…. EXACTLY. Are you ready to review now?

1)  How do these patients present?

HISTORY, HISTORY, HISTORY. The classic example goes something like: A family of five presents from home in the dead of winter complaining of headaches associated with a myriad of vague, nonspecific complaints.

Later on, you overhear mom mention, out of frustration, that she had to pay for an Uber XL to get the whole fam to the ED tonight, since dad’s Camero was ‘on empty.’   Turns out, upon arriving home from the bars last night, dad accidentally left his car keys in the ignition before passing out in the doorway connecting the garage to the kitchen. (Ok, so maybe you won’t see that last part on the boards, but it’s my favorite real-life example).

The most common complaints: (1) HEADACHE, (2) nausea, (3) dizziness, and mental status changes with more severe toxicity (memory disturbances commonly manifested as amnesia, decreased cognition, stupor, coma, gait disturbances, etc). Keep in mind that the list of potential associated complaints is broad and encompass nearly every organ system.

Don’t count on the physical exam to nail the diagnosis. Remember that pulse oximetry is not affected. Know the traditional buzz words “cherry red” lips and skin for the boards; but also know that these are rarely seen in clinical practice.

2) How is it diagnosed?

Send a co-oximetry panel. Don’t get tripped up on details- you can send either a venous or arterial blood sample. If your clinical suspicion is high, do not delay treatment pending results.

A CO level >3% in non-smokers, or >10% in smokers, is diagnostic.

The actual percentages weakly correlate with associated symptoms and overall prognosis.  That being said, in the proper clinical setting, you can make the diagnosis and treat presumptively with normal or borderline CO levels.

3) How is it treated?

ABC’s- Intubate if the patient is altered and unable to protect his/her airway. Administer 100% oxygen via NRB. Keep this patient on a cardiac monitor. If for no other reason, CO binds to cardiac myoglobin with an even greater affinity than to hemoglobin, resulting in cardiac ischemia, ventricular arrhythmias, and cardiovascular collapse in severe cases. Finally, know your indications for Hyperbaric Oxygen (HBO).**

**note: this list is variable (and debatable) depending on the source, but generally accepted indications include: anyone who is pregnant, anyone who has signs of cardiac ischemia, history of prolonged LOC, or presence of neurological deficits.

  • Focal neurological deficits, coma, h/o transient LOC (transient LOC = independent risk factor for increased morbidity)
  • Pregnancy (with CO > 15%)
  • Evidence of cardiac ischemia, usually on EKG (or h/o CAD with CO >20%)
  • Basically any symptoms with CO >40%
  • Symptoms that don’t resolve after 6 hrs of 100% O2 via NRB

KEEP IN MIND: Clearance of CO via:

Room air: ~300 minutes

100% NRB: ~90 minutes

HBO: ~15-30 minutes

IDEALLY, TRANSFER FOR HBO SHOULD BE MADE ON A CASE-BY-CASE BASIS, AND SHOULD TAKE INTO CONSIDERATION THE STABILITY OF THE PATIENT FOR TRANSFER AND THE TIME INVOLVED FOR THE TRANSFER PROCESS ITSELF, AMONG OTHER THINGS.

The reason we transfer patients for HBO therapy = prevention of long-term neurologic sequelae.  

 

Want to learn more?

https://lifeinthefastlane.com/ccc/carbon-monoxide-poisoning/

https://lifeinthefastlane.com/ccc/hyperbaric-oxygen-and-carbon-monoxide-poisoning/

https://emedicine.medscape.com/article/819987-treatment#d12

https://emcrit.org/racc/cardiac-arrest-after-smoke-inhalation/

 

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