PODT: Urushiol-Producing Plants

In North America, contact dermatitis from plant exposure is most commonly caused by poison ivy, oak, and sumac.

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These plants secrete urushiol oil, which causes a type IV hypersensitivity reaction (cell-mediated, delayed) when in contact with the skin, GI tract, or respiratory tract (through smoke).

Patients often present days to weeks post-exposure, as the oil can stay on clothes, equipment, pets, etc. for up to years. As such, it is important to advise patients to thoroughly wash everything that may have been exposed ASAP. After exposure, it takes approximately 20-30 minutes for urushiol to penetrate the skin, so washing skin as soon as possible after a possible exposure can prevent reaction.

Rash is vesicular, pruritic, and linear. Appears 1-2 days post-exposure (possibly shorter if patient has prior exposure, longer if naïve).

The vesicles seen above contain clear fluid that is not contagious. Patients may say the rash is “spreading,” but they likely have just not fully decontaminated and still transferring oil to other parts of their body.

The vesicles seen above contain clear fluid that is not contagious. Patients may say the rash is “spreading,” but they likely have just not fully decontaminated and still transferring oil to other parts of their body.

Treatment is generally supportive. Symptoms can be treated with oral antihistamines, calamine lotion, oatmeal baths, cool compresses, topical astringents under occlusion dressings to dry weeping lesions.

For severe reactions, 15-20 day steroid taper can be prescribed. Low-dose steroid bursts are not recommended, as rebound dermatitis is seen.

Not indicated are topical antihistamines, anesthetics containing benzocaine, antibiotics containing neomycin or bacitracin.

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POTD: ABG vs VBG

Today we look at the difference between ABG and VBG in the ED clinical setting: When should you order an ABG and when does a VBG suffice?

pH

-        Correlates closely

-        VBG typically 0.03-0.04 lower than ABG

pCO2

-        VBG can be used as a screen for hypercarbia (100% sensitive) for values < 45 mmHg

-        ABG should be used to assess pCO2 for patients in shock states and when value is > 45 mmHg

-        Mean difference is PvCO2 = PaCO2 + 5.7 mmHg

PO2

-        Does not correlate

-        ABG should be collected depending on clinical circumstance, ie. ARDS, severe hypoxemia, severe acidosis, poor peripheral perfusion, pulse ox not accurate, etc.

HCO3

-        Correlates closely

-        Is a calculated value - get a BMP for more accurate result

Lactate

-        Correlates closely

-        Mean difference is 0.02-0.08

Base Deficit / Base Excess

-        Correlates closely

-        Difference is not clinically significant

 

Remember:

-        ABGs are not without risk!

-        Disadvantages include risk of bleeding/hematoma, pain, nerve injury, digital ischemia, delays in care, risk of pseudoaneurysm and AV fistula, etc.

 

Takeaways:

-        Get an ABG in patients with severe shock, hypoxemia, or when PaO2 is clinically relevant.

-        A VBG is sufficient to trend pH, lactate, pCO2 when not hypercarbic, and base deficit.

-        If you’re really concerned about HCO3, check a BMP.

-        As always, any test you order should be clinically relevant and benefits outweigh the risks.

 

More reading:

https://litfl.com/vbg-versus-abg/

https://epmonthly.com/article/blood-gases-abg-vs-vbg/

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