Deep Neck Space Infections, AHHH!

 ·   · 
WELCOME ONE AND ALL TO THE GLORIOUS DOROS ADMIN MONTH!

 

Today we will be talking about DEEP SPACE NECK INFECTIONS , because they are doozies. Usually these get mis-triaged to SPLITFLOW, so for new 2nd years, be careful!

 

THE BASICS

  • The neck/pharynx is one of three potential spaces that can cause deep neck space infections
    1. retrophanygeal space
    2. submandibular space
    3. parapharyngeal space
  • Usually are PolymicrobialImage result for deep space neck infections em

     

     

     

    HOW DO THEY PRESENT??? (The starred ones are really concerning)

  • Trismus *******
  • Swelling below angle of mandible
  • Lymphadenopathy
  • Poor handling of oral secretions ******
  • Asymetric pharyngeal swelling
  • Odynophagia (painful swallowing)
  • Muffuled voice ***** 

     

     

    Differential?

    Based on the symptoms, concern

     

    • Peritonsillar Abscess, Retropharyngeal Abscess
    • Ludwig's Angina
    • Epiglottitis
    • Lymphoma
    • Lemierre's Syndrome
    • Parapharyngeal abscess
    • LIST GOES ON!!!!

     

    BUT JOSH!?? HOW DO WE DIAGNOSE THESE????

    IMAGING IMAGING IMAGING

    When in doubt, get a CT with IV contrast to evaluate for deep neck space infections. However there are some signs on Neck Soft Tissue XR. Look for widening of the pre-vertebral spaces.

    Image result for retropharyngeal abscess x ray

     

     

     

    TREATMENT?????

    AIRWAY AIRWAY AIRWAY, then maybe some ABX/Drainage

    If you think that their airway is compromised, get all your airway equipment ready, monitor them, get ENT on board early, intubate early, possibly in the OR with ENT.

     

     

    TAKEAWAY POINTS:

    If you have someone in SPLIT with muffled voice, 1 finger trismus, with difficulty swallowing these patients, upgrade them immediately, watch their airway, get imaging and consultants on board early, and DON'T PANIC (or do.....) 

     

     

     

 · 
Share

Respiratory fRiday: Platypnea, platypus, orthodeoxia, orthodontics?

Platypnea-orthodeoxia:
* What's that you say? 
dyspnea and deoxygenation when moving to SITTING UPRIGHT from supine
= wait, that's not normal!
= improved with lying FLAT
- due to INTRAPULMONARY shunting when moving UPRIGHT
- suspect with:
hepatopulmonary syndrome
- possible mechanism of vasodilation and thus increased perfusion of capillaries at lung BASES (which are least oxygenated)
- liver cirrhosis- see this in ~40% of cirrhotic patients
- portal HTN
- hepatitis
- look for finger clubbing!
- pulmonary congestion at bases
AV shunting
ASD or PFO w/ shunting
ARDS = rare causes
- Pericardial effusion/restrictive pericarditis
- Significant PE load
- Ileus
* Why do I care? 
- Treatment in the ER: 
- Increased mortality in cirrhotic patients especially with PO2 <60mmHg
LIVER TRANSPLANT: definitive (but lower post-transplant survival)
TIPS: decrease shunting, improved gas exchange (theoretical)
- Nitric Oxide Synthesis inhibition or TNF inhibition: no evidence yet
 
- MDM:
not all respiratory distresses = CHF, pneumonia or PE
- consider extra-pulmonary contribution to SOB
not all treatments will work all the time
- keep an eye out for variation and be suspicious something else is going on
 · 
Share

Toxic Thursday: EKG changes in overdoses!

To follow-up on the digoxin toxicity POD from aggggggges ago, here's a sampling of some pathognomonic EKG changes in the setting of drug toxicity (this is NOT all-encompassing-- there's a lot more out there!) 1) Older woman with HTN comes in w/ dizziness. 2) Young disheveled guy, somnolent. 3) Old guy on blood thinner w/ HTN, new renal failure and weakness. ​

4) Old Asian man with cardiac and renal problems comes in vomiting (1st EKG from PMD's office, 2nd from MMC)

5) Old lady with vomiting syncopizes (First EKG progresses to second

​ ------------

Answers:

1) Long PR interval = AV nodal delay

= 1st degree Heart Block (may progress to complete heart block)

- Beta-blockade

Calcium-channel blockade

Digoxin

Opioids, clonidine

2) Interventricular slowed conduction w/ long QRSright axis deviation w/ R/S ration >0.7 in aVR

Na-channel blockade

TCA toxicity

- Anticholinergics

Na-channel blockers like the "ides'

- Propanolol

- Anesthetics like bupivicaine

- Carbamazepine

 

3) Atrial tachycardia w/ AV block and PVCs (can also have PACs)

digoxin! (classical finding)

 

4) Ventricular dysrhythmia w/ beat-to-beat alteration of QRS orientation

Bidirectional Ventricular Tachycardia  (may also have multiple ectopic beats mixed in)

- digoxin! (pathognomonic finding)***

- also a few herb toxidromes (aconite)

​ 5) Long QT = prolongation of repolarization = risk of Torsades

- Non-toxicological risks: female, >60yo, genetics, structural heart dz/LV dysfunction

Sympathicomimetics and:

​- Use the QT normogram (based on absolute QT) to predict risk of Torsades! (drugs that cause bradycardia are MORE likely to cause Torsades)

*** Just to summarize, the most common EKG presentations of digoxin toxicity are:

1) PACs/PVCs

2) Atrial tachycardia w/ AV block (classic)

3) Bidirectional VT (answer on tests)

 

 · 
Share